R&D
1 The nonspecific immune suppression by TNF-α inhibitor
2 one-third of patients do not respond adequately to TNF-α inhibitors
3 some patients may respond initially but lose sensitivity over time
4 increased incidence of infections like tuberculosis, bacterial infections, septic arthritis, and opportunistic infections
5 high cost of the biologics
The peptide has superior HDAC inhibition and at the same time, it increases significant chondrogenic activity, as seen in the strong blue alcian staingn compared to anti-TN-alpha antibody. As a result, arthritic animal has been completely healed by the peptide to reach to the normal cartilage level, which is also contrast to anti-TNF-alpha antibody.